Publication | Open Access

Age exaggerates proinflammatory cytokine signaling and truncates signal transducers and activators of transcription 3 signaling following ischemic stroke in the rat

InflammationAgingIschemic StrokeStrokeSignal TransducersMedicineImmunologyCerebrovascular DiseaseVascular BiologyNeurologyTranscription 3Cerebral Blood FlowNeuroimmunologyCell BiologyAtherosclerosisIschemic SyndromeCerebral Vascular RegulationNeuroinflammation