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Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucose

Signal TransductionBiochemistryG Protein-coupled ReceptorMedicineReceptor Tyrosine KinaseDiabetesInsulin ManagementInsulin DeliveryEndocrinologyPharmacologyCell SignalingInsulin Signaling