Publication | Open Access

Mitochondria-targeted drugs enhance Nlrp3 inflammasome-dependent IL-1β secretion in association with alterations in cellular redox and energy status

InflammationRedox SignalingAnti-inflammatoryMitochondrial FunctionMedicineImmunologyChronic InflammationCellular RedoxInflammasomeEnergy StatusMitochondria-targeted DrugsPharmacologyCell BiologyCell SignalingOxidative Stress