ZEB1 overexpression associated with E-cadherin and microRNA-200 downregulation is characteristic of undifferentiated endometrial carcinoma

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The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

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A reciprocal repression between ZEB1 and members of the miR‐200 family promotes EMT and invasion in cancer cells

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The EMT-activator ZEB1 promotes tumorigenicity by repressing stemness-inhibiting microRNAs

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Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like Phenotype

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