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miR-21 overexpression enhances TGF-β1-induced epithelial-to-mesenchymal transition by target smad7 and aggravates renal damage in diabetic nephropathy

Mir-21 OverexpressionRenal PathologyRenal InflammationRenal DamageTarget Smad7Kidney Tubule RemodelingChronic Kidney DiseaseCell SignalingVascular BiologyRenal PathophysiologyMicrorna DetectionCell BiologyUrologyRenal DiseaseDiabetesDiabetic Kidney DiseaseMedicineNephrologyKidney Research