Publication | Closed Access

Deletion of the last five C-terminal amino acid residues of connexin43 leads to lethal ventricular arrhythmias in mice without affecting coupling via gap junction channels

Gap Junction ChannelsMolecular PharmacologyVentricular ArrhythmiasCardiomyopathyMolecular PhysiologyCardiac MuscleMedicineSynaptic TransmissionPhysiologyIon ChannelsNeuropharmacologyElectrophysiologyPharmacologyConnexin43 Leads