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Implications of the Progressive Self-association of Wild-type Human Factor H for Complement Regulation and Disease

HistocompatibilityComplement SystemProgressive Self-associationAutoimmune DiseaseDisease MechanismGeneticsComplement RegulationImmunologyHematologyGenetic EpidemiologyAutoimmunitySelf-toleranceHla TypingMedicineImmune-related Gene Polymorphism