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Integration of pharmacokinetic and NRF2 system biology models to describe reactive oxygen species production and subsequent glutathione depletion in liver microfluidic biochips after flutamide exposure

BiochemistrySubsequent Glutathione DepletionMedicineLiver PhysiologyPhysiologyBioanalysisLipid PeroxidationToxicologyHepatotoxicityFlutamide ExposureReactive Oxygen SpecieMetabolomicsMetabolismLiver Microfluidic BiochipsRedox BiologyDrug-induced Liver InjuryOxidative StressHealth Sciences