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Phenotypic anchoring of arsenic and cadmium toxicity in three hepatic-related cell systems reveals compound- and cell-specific selective up-regulation of stress protein expression: Implications for fingerprint profiling of cytotoxicity

Cadmium ToxicityCell DeathToxicological MechanismPhenotypic AnchoringOxidative StressToxicologyHepatotoxicityCell SignalingBiochemistryLiver PhysiologyStress Protein ExpressionExperimental ToxicologyGene ExpressionCell BiologyNatural SciencesForensic ToxicologyMetal ToxicitySystems BiologyMedicineToxicogenomics