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Genetic deletion of the adenosine A2A receptor in mice reduces the changes in spinal cord NMDA receptor binding and glucose uptake caused by a nociceptive stimulus

Health SciencesMedicinePhysiologyNeuropathic PainNeuropeptide ReceptorNeuropharmacologyMolecular PainNeurosciencePain MechanismMolecular NeurobiologyNervous SystemPharmacologyGlucose UptakeAdenosine A2a ReceptorGenetic DeletionPain Research