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Suppression of NF-κB signaling and NLRP3 inflammasome activation in macrophages is responsible for the amelioration of experimental murine colitis by the natural compound fraxinellone

InflammationChronic Inflammatory DiseasesAnti-inflammatoryMucosal ImmunologyMicrobial PathogensExperimental Murine ColitisImmunologyImmune RegulationChronic InflammationInflammation BiologyPathologyNatural Compound FraxinelloneInnate ImmunityMedicineInflammatory DiseaseNf-κb Signaling