Publication | Open Access

Cardiomyocyte-specific expression of CRNK, the C-terminal domain of PYK2, maintains ventricular function and slows ventricular remodeling in a mouse model of dilated cardiomyopathy

Cardiac MuscleCardiomyopathyHeart FailureMolecular PhysiologySignal TransductionCell SignalingCardiomyocyte-specific ExpressionPhysiologyMouse ModelCardiovascular PhysiologyMedicineCell BiologyCardiologyCellular PhysiologyC-terminal Domain