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Chronic treatment of cultured cerebral vascular smooth cells with low concentration of ethanol elevates intracellular calcium and potentiates prostanoid-induced rises in [Ca2+]i: Relation to etiology of alcohol-induced stroke

ThrombosisNeurophysiologyMedicinePharmacologyCerebrovascular DiseaseVascular BiologyNeuroscienceNeurologyAlcohol-induced StrokeCerebral Blood FlowReperfusion InjuryStrokeLow ConcentrationAtherosclerosisIschemic SyndromeSocial SciencesCerebral Vascular RegulationChronic Treatment