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Compound K, an intestinal metabolite of ginsenosides, inhibits PDGF-BB-induced VSMC proliferation and migration through G1 arrest and attenuates neointimal hyperplasia after arterial injury

InflammationAngiogenesisG1 ArrestMedicineCompound KEndothelial DysfunctionArterial InjuryPathologyVascular BiologyNeovascularizationPharmacologyCell BiologyCell Signaling