The Neurotoxicity Induced by Ethanol Withdrawal in Mature Organotypic Hippocampal Slices Might Involve Cross‐Talk Between Metabotropic Glutamate Type 5 Receptors and <i>N</i>‐Methyl‐d‐Aspartate Receptors

Abstract

From this model of long-term ethanol exposure and withdrawal, the functional interplay between mGluR5s and NMDARs might represent a novel target for the prevention of neurotoxicity associated with ethanol withdrawal.