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Comparison of mice deficient in the high- or low-affinity neurotensin receptors, Ntsr1 or Ntsr2, reveals a novel function for Ntsr2 in thermal nociception

Low-affinity Neurotensin ReceptorsNeurophysiologyMedicinePhysiologyNeuropathic PainMice DeficientNeuropharmacologyNeuropeptide ReceptorSocial SciencesNeurosciencePain MechanismPharmacologyThermal NociceptionSensationNeuropeptidesPain Research