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Reactive oxygen species generation and mitochondrial dysfunction in the apoptotic cell death of human myeloid leukemia HL-60 cells by a dietary compound withaferin A with concomitant protection by N-acetyl cysteine

N-acetyl CysteineMitochondrial FunctionBiochemistryMitochondrial DysfunctionMedicineApoptosisLipid PeroxidationCell DeathReactive Oxygen SpecieDietary CompoundMetabolismPharmacologyCell BiologyRedox BiologyOxidative Stress