Analysis of gene expression in human bronchial epithelial cells upon influenza virus infection and regulation by p38 mitogen‐activated protein kinase and c‐Jun‐N‐terminal kinase

Abstract

Although further studies are needed to obtain a more complete understanding of gene expression and the role of MAPK in gene expression, the present results are important in understanding the molecular mechanisms involved in the response of bronchial epithelial cells to IV infection.