Memory loss caused by β-amyloid protein is rescued by a β3-adrenoceptor agonist

Diffusible, nonfibrillar ligands derived from Aβ _1–42 are potent central nervous system neurotoxins

Mary P. Lambert, Avlin Barlow, Brett A. Chromy et al. · Proceedings of the National Academy of Sciences · 1998 · 3.5K citations · Full text

Natural Oligomers of the Alzheimer Amyloid-β Protein Induce Reversible Synapse Loss by Modulating an NMDA-Type Glutamate Receptor-Dependent Signaling Pathway

Ganesh M. Shankar, Brenda L. Bloodgood, Matthew Townsend et al. · Journal of Neuroscience · 2007 · 1.6K citations · Full text

Mutant presenilins specifically elevate the levels of the 42 residue β-amyloid peptide in vivo: evidence for augmentation of a 42-specific γ secretase

Joanna L. Jankowsky, Daniel J. Fadale, Jeffrey C. Anderson et al. · Human Molecular Genetics · 2003 · 1.6K citations · Full text

Independent Cellular Processes for Hippocampal Memory Consolidation and Reconsolidation

Jonathan Lee, Barry J. Everitt, Kerrie L. Thomas · Science · 2004 · 819 citations

Memory Retrieval, Neuropsychology, Structural Plasticity +17

Detection of Abnormal Memory Decline in Mild Cases of Alzheimer's Disease Using CERAD Neuropsychological Measures

Katherine M. Welsh, Nelson Butters, James P. Hughes et al. · Archives of Neurology · 1991 · 732 citations

Neuropsychology, Mild Cases, Cerad Patients +21