Publication | Open Access

A copper(II)-selective chelator ameliorates diabetes-evoked renal fibrosis and albuminuria, and suppresses pathogenic TGF-β activation in the kidneys of rats used as a model of diabetes

InflammationDiabetes-evoked Renal FibrosisUrologyRenal FunctionSodium HomeostasisMedicineBioactive MetalDiabetesPhysiologyPharmacologyRenal InflammationDiabetic Kidney DiseaseRenal PathophysiologyPathogenic Tgf-β ActivationChronic Kidney DiseaseNephrologyKidney Research