Publication | Open Access

Variations in the protein level of Omi/HtrA2 in the heart of aged rats may contribute to the increased susceptibility of cardiomyocytes to ischemia/reperfusion injury and cell death

Metabolic SyndromeCardiomyopathyHeart FailureAgingCardiovascular DiseaseAged RatsPhysiologyCell DeathProtein LevelCardiologyCardiovascular FunctionCardiovascular ToxicityMedicineCell BiologyAtherosclerosisOxidative StressHealth Sciences