Publication | Open Access

Human lipodystrophies linked to mutations in A-type lamins and to HIV protease inhibitor therapy are both associated with prelamin A accumulation, oxidative stress and premature cellular senescence

Premature Cellular SenescenceBiochemistryA-type LaminsDegenerative PathologyCytoskeletonLipoprotein MetabolismHivMedicineCell BiologyOxidative Stress