Preferential sites for the integration and disruption of human papillomavirus 16 in cervical lesions

Cancer Incidence in Five Continents

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J. Virol. Methods

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Adenovirus E1A, simian virus 40 tumor antigen, and human papillomavirus E7 protein share the capacity to disrupt the interaction between transcription factor E2F and the retinoblastoma gene product.

Srikumar Chellappan, Virginia B. Kraus, Burkhard Kröger et al. · Proceedings of the National Academy of Sciences · 1992 · 624 citations · Full text

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Integration of human papillomavirus type 16 DNA into the human genome leads to increased stability of E6 and E7 mRNAs: implications for cervical carcinogenesis.

Saewha Jeon, Paul F. Lambert · Proceedings of the National Academy of Sciences · 1995 · 539 citations

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The human papillomavirus type 16 E6 and E7 oncoproteins cooperate to induce mitotic defects and genomic instability by uncoupling centrosome duplication from the cell division cycle

Stefan Duensing, Lily Y. Lee, Anette Duensing et al. · Proceedings of the National Academy of Sciences · 2000 · 493 citations

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