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Vitamin C prevents stress-induced damage on the heart caused by the death of cardiomyocytes, through down-regulation of the excessive production of catecholamine, TNF-α, and ROS production in Gulo(−/−) mice

Cardiac MuscleCardiomyopathyCardiovascular DiseaseMedicinePhysiologyCardiometabolic PhysiologyCell DeathVitamin CStress-induced DamageExcessive ProductionCardiovascular FunctionCardiovascular ToxicityPharmacologyCell BiologyCardiologyOxidative Stress