Publication | Open Access

Low levels of selenium compounds are selectively toxic for a human neuron cell line through ROS/RNS increase and apoptotic process activation

Apoptotic Process ActivationSelenium CompoundsBiochemistrySelenium DeficiencyMedicineLipid PeroxidationToxicologyNeuroscienceReactive Oxygen SpecieExperimental ToxicologyPharmacologyToxicological MechanismOxidative StressRos/rns Increase