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Effects of copper overload in P19 neurons: impairment of glutathione redox homeostasis and crosstalk between caspase and calpain protease systems in ROS-induced apoptosis

Reductive StressRedox SignalingSignal TransductionBiochemistryCopper OverloadApoptosisPhysiologyGlutathione Redox HomeostasisCell DeathP19 NeuronsNeuroprotectionRedox BiologyReactive Oxygen SpecieMedicineCell BiologyCell SignalingOxidative Stress