<i>In vivo</i> characterization of the inflammatory infiltrate and apoptotic status in imiquimod‐treated basal cell carcinoma

Abstract

Our results support the hypothesis that imiquimod activity in the treatment of BCC is partly a result of a pro-inflammatory action mediated by CD3(+)/CD4(+) lymphoid cells and of a pro-apoptotic activity associated with decreased Bcl-2 expression.