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Mechanism of impairment of long-term potentiation by amyloid β is independent of NMDA receptors or voltage-dependent calcium channels in hippocampal CA1 pyramidal neurons

Amyloid βSynaptic PlasticityNeurodegenerative DiseasesAlzheimer's DiseaseNeurophysiologySynaptic TransmissionPhysiologyNmda ReceptorsLong-term PotentiationNeuroscienceNeurotransmissionNeurodegenerationMolecular NeurobiologyMedicineSynaptic SignalingSocial Sciences