Publication | Open Access

Cold tolerance of UCP1-ablated mice: A skeletal muscle mitochondria switch toward lipid oxidation with marked UCP3 up-regulation not associated with increased basal, fatty acid- or ROS-induced uncoupling or enhanced GDP effects

Energy MetabolismMolecular PhysiologyMitochondrial FunctionBiochemistryPhysiologyGdp EffectsMetabolic RegulationUcp1-ablated MiceMetabolismMedicineCell BiologyCellular PhysiologyMarked Ucp3 Up-regulationOxidative StressHealth Sciences