<scp>N</scp>‐acetyl‐<scp>L</scp>‐cysteine protects against cadmium‐induced neuronal apoptosis by inhibiting<scp>ROS</scp>‐dependent activation of<scp>A</scp>kt/m<scp>TOR</scp>pathway in mouse brain

Abstract

NAC protects against Cd-induced neuronal apoptosis in mouse brain partially by inhibiting ROS-dependent activation of Akt/mTOR pathway. The findings highlight that NAC may be exploited for prevention and treatment of Cd-induced neurodegenerative diseases.