Publication | Open Access
Effects of the novel (Pro3)GIP antagonist and exendin(9–39)amide on GIP- and GLP-1-induced cyclic AMP generation, insulin secretion and postprandial insulin release in obese diabetic (ob/ob) mice: evidence that GIP is the major physiological incretin
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Citations
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References
2003
Year
Metabolic SyndromeInsulin SignalingBiochemistryMedicineInsulin ManagementPhysiologyDiabetesGip AntagonistMetabolic RegulationEndocrinologyMetabolismInsulin SecretionMetabolic SignalingPharmacologyMajor Physiological IncretinGastrointestinal Peptide HormoneHealth Sciences
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