PANCREATIC INSUFFICIENCY AND HYPERPARATHYROIDISM

Abstract

Case Reports1 September 1957PANCREATIC INSUFFICIENCY AND HYPERPARATHYROIDISMIRVIN C. PLOUGH, LAURENCE H. KYLE, M.D.IRVIN C. PLOUGHSearch for more papers by this author, LAURENCE H. KYLE, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-47-3-590 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThe most frequent cause of hyperparathyroidism is an adenoma of a parathyroid gland. The course of events leading to adenoma formation in the parathyroids, as in other tissues of the body, is unknown. In some organs, conditioning factors can be identified which seem to predispose to the formation of such tumors. For example, in the thyroid gland, iodine deficiency produces hyperplasia and nodule formation, and on occasion functionally active adenomas appear. The possibility has been considered that a similar train of events sometimes precedes the appearance of a parathyroid adenoma.1In conditions such as rickets, osteomalacia and renal insufficiency, where...Bibliography1. AlbrightReifenstein FEC: The parathyroid glands and metabolic bone disease, 1948, The Williams and Wilkins Co., Baltimore, p. 46. Google Scholar2. HowardHopkinsConnor JETRTB: On certain physiologic responses to intravenous injection of calcium salts into normal, hyperparathyroid and hypoparathyroid persons, J. Clin. Endocrinol. 13: 1, 1953. CrossrefMedlineGoogle Scholar3. KyleSchaafErdman LHMLA: The metabolic effects of intravenous administration of calcium, J. Lab. and Clin. Med. 43: 123, 1954. MedlineGoogle Scholar4. NordinFraser BER: The effect of intravenous calcium on phosphate excretion, Clin. Sc. 13: 477, 1954. MedlineGoogle Scholar5. PloughKyle ICLH: Pseudofractures with hypercalcemia: osteomalacia vs. hyperparathyroidism (abstract), J. Clin. Endocrinol. 15: 870, 1955. Google Scholar6. AlbrightReifenstein FEC: The parathyroid glands and metabolic bone disease, 1948, The Williams and Wilkins Co., Baltimore, p. 109. Google Scholar7. Dent CE: Personal communication. Google Scholar8. AlbrightReifenstein FEC: The parathyroid glands and metabolic bone disease, 1948, The Williams and Wilkins Co., Baltimore, p. 262. Google Scholar9. Lynch MJ: Nephrosis and fat embolism in acute hemorrhagic pancreatitis, Arch. Int. Med. 94: 709, 1954. CrossrefGoogle Scholar10. Gutman AB: Primary and secondary gout, Ann. Int. Med. 39: 1062, 1953. LinkGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: *Received for publication April 18, 1956.From the Department of Metabolism, Medical Division, Army Medical Service Graduate School, and the Department of Medicine, Walter Reed Army Hospital, Walter Reed Army Medical Center, Washington, D. C.Requests for reprints should be addressed to Irvin C. Plough, Lt. Colonel (MC) USA, Department of Metabolism, Walter Reed Army Institute of Research, Walter Reed Army Medical Center, Washington 12, D. C. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byChronic pancreatitis in primary hyperparathyroidism: Comparison with alcoholic and idiopathic chronic pancreatitisPrimary hyperparathyroidismPANCREATITIS AND PRIMARY HYPERPARATHYROIDISM: FORTY CASESParathyroid crisis and necrotising pancreatitisErnährungs- und stoffwechselbedingte Osteopathien bei ErwachsenenErnährungs- und stoffwechselbedingte Osteopathien bei ErwachsenenParathyroid Physiology and Primary HyperparathyroidismPancreatitis and hyperparathyroidismPathophysiology of the Exocrine PancreasPankreaserkrankungen bei Erkrankungen anderer OrganeFamilial chronic pancreatitis associated with pancreatic lithiasisPANCREATITIS ASSOCIATED WITH HYPERPARATHYROIDISMSome Metabolic Aspects of Exocrine Pancreatic DiseasePancreatic Insufficiency as the Presenting Feature of HyperparathyroidismANDREW L. WARSHAW, M.D., WILLIAM D. HEIZER, M.D., LEONARD LASTER, M.D., F.A.C.P.“Tertiary” hyperparathyroidism induced by osteomalacia resulting from phosphorus depletionAcute arthritisPancreatitis Associated with Primary HyperparathyroidismUric acid, calcium and phosphorus clearances in normal subjects on a low calcium, low phosphorus diet: Uric acid, calcium and phosphorus clearances after calcium infusion in normal and gouty patientsCoexistence of Pancreatitis and Hyperparathyroidism Use of the Secretin Test in Evaluation of Pancreatic Function before and after Excision of a Parathyroid AdenomaD. J. KURLANDER, M.D., H. F. RASKIN, M.D., J. B. KIRSNER, M.D., PH.D.Acute Pancreatitis Secondary to Hypercalcemia of Multiple MyelomaLAWRENCE E. MELTZER, M.D., FLORENTINO P. PALMON JR., M.D., YOUNG K. PAIK, M.D., R. PHILIP CUSTER, M.D.Pancreatitis associated with hyperparathyroidismFurther Experience with Pancreatitis as a Diagnostic Clue to HyperparathyroidismHyperuricemia in HyperparathyroidismRecurrent Acute Hyperparathyroidism with Severe Gastrointestinal ManifestationsThe PancreasSOME RECENT DEVELOPMENTS PERTAINING TO PANCREATITIS*JOHN B. GROSS, M.D.HEREDITARY HYPERPARATHYROIDISM ASSOCIATED WITH RECURRENT PANCREATITIS*†CHARLES E. JACKSON, M.D., F.A.C.P. 1 September 1957Volume 47, Issue 3Page: 590-598KeywordsAdenomasGraduate medical educationHospital medicineHyperparathyroidismMedical servicesPancreasPancreatitisParathyroidThyroidThyroid carcinoma ePublished: 1 December 2008 Issue Published: 1 September 1957 PDF downloadLoading ...