• Thrombosis is framed as a central driver of arterial atherosclerosis, with studies linking clot formation to progressive arterial disease and occlusion across coronary and systemic vessels [6], [10], [12], [14].

• Coagulation physiology and hereditary coagulopathies are depicted as key determinants of thrombosis risk, integrating experimental clotting factors, antihemophilic interventions, and familial traits into vascular disease models [2], [9], [18], [20].

• Carotid and cerebrovascular occlusion are characterized through angiographic and anatomical studies, highlighting spontaneous thrombosis, carotid artery occlusion, and diagnostic imaging patterns [1], [5], [11].

• Coronary disease progression and occlusion mechanisms are explored via prognostic courses, arteriosclerotic changes, and occlusion physiology shaping myocardial outcomes [4], [7], [8], [10].

• Microvascular structure and venous endothelium context are linked to thrombosis via endothelial responses to flow, venous injury, and capillary physiology, informing the vascular thrombosis landscape [13], [16], [17].