Bivalent Chromatin Mark Analysis is a methodological framework in epigenomics that identifies and characterizes genomic regions co-marked by both activating (H3K4me3) and repressive (H3K27me3) histone modifications to delineate developmentally regulated, poised transcriptional states in chromatin.
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Publications
176
Citations
19
Authors
9
Institutions
Publications per year
2017–2026
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Publications
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Hdac3, Setdb1, and Kap1 mark H3K9me3/H3K14ac bivalent regions in young and aged liver
Andrew J. Price, Mohan C. Manjegowda, Jessica Kain et al. · Aging Cell · 2019 · 53 citations · Full text
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Authors
19
- JW
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- HC
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- JK
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- IM
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- AH
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1–5 of 19
Institutions
9
Max Planck Institute for Molecular Genetics
Berlin, Germany
1 h-index
4 publications
412 citations
Epigenomics (Germany)
Berlin, Germany
1 h-index
4 publications
412 citations
University of Pennsylvania
Philadelphia, United States
1 h-index
4 publications
80 citations
University of Virginia
Charlottesville, United States
1 h-index
5 publications
265 citations
Saarland University
Saarbrücken, Germany
1 h-index
2 publications
206 citations
Max Delbrück Center
Berlin, Germany
1 h-index
2 publications
206 citations
Charité - Universitätsmedizin Berlin
Berlin, Germany
1 h-index
2 publications
206 citations
Children's Hospital of Philadelphia
Philadelphia, United States
1 h-index
2 publications
40 citations
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Venues
- Nature Communications
1 publications · 103 citations
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- Aging Cell
1 publications · 53 citations
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- Cells
1 publications · 20 citations
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- Toxicology in Vitro
1 publications · 10 citations
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