Nathan M. Appel - Concepedia

Author

Nathan M. Appel

National Institutes of Health

Also Known As

NATHAN M APPEL, NATHAN M. APPEL, Nathan Appel, Nathan M Appel, Nathan M. Appel

Publications

1.6K

Citations

H-Index

Concepts

All Affiliations

National Institutes of Health National Institute on Aging Center for Drug Evaluation and Research University of Toronto United States Food and Drug Administration

About

Nathan M. Appel is an author at National Institutes of Health specializing in medicine, neuroscience, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	18	1K
2	Health Sciences	6	6	408
3	Engineering	2	2	53
4	Natural Sciences	1	1	15
5	Public Health	1	1	15

Nathan M. Appel

Publications

1.4K

Citations

H-Index

Page 1

	Year	Citations
Blood—Brain Barrier Glucose Transporter Ian A. Simpson, Nathan M. Appel, Mitsuhiko Hokari, Journal of Neurochemistry Glut1 Glucose TransportersGlycobiologyOptogeneticsInsulin SignalingSocial Sciences	1999	301
The Consortium of Metabolomics Studies (COMETS): Metabolomics in 47 Prospective Cohort Studies Bing Yu, Krista A. Zanetti, Marinella Temprosa, American Journal of Epidemiology	2019	106
β-Endorphin-Induced Stimulation of Central Sympathetic Outflow: β-Endorphin Increases Plasma Concentrations of Epinephrine, Norepinephrine, and Dopamine in Rats* Glen R. Van Loon, Nathan M. Appel, Doris Ho Endocrinology PharmacotherapyCentral Sympathetic OutflowSympathetic Nervous SystemNeuroendocrine MechanismPlasma Dopamine	1981	104
Thyrotropin-releasing hormone in spinal cord: coexistence with serotonin and with substance P in fibers and terminals apposing identified preganglionic sympathetic neurons Nathan M. Appel, Martin W. Wessendorf, Robert Elde Brain Research NeurophysiologyNeuroanatomySympathetic Nervous SystemPhysiologyNeuroendocrine Mechanism	1987	104
β-Endorphin-induced hyperglycemia is mediated by increased central sympathetic outflow to adrenal medulla Glen R. Van Loon, Nathan M. Appel Brain Research Metabolic Syndromeβ-Endorphin-induced HyperglycemiaAdrenal GlandPhysiologyDiabetes	1981	71
Mu receptors at discrete hypothalamic and brainstem sites mediate opioid peptide-induced increases in central sympathetic outflow Nathan M. Appel, Judith A. Kiritsy‐Roy, Glen R. Van Loon Brain Research NeuropeptidesNeurophysiologyPhysiologyNeuroendocrine MechanismHypothalamic Peptide	1986	62
Effects of high‐dose fenfluramine treatment on monoamine uptake sites in rat brain: Assessment using quantitative autoradiography Nathan M. Appel, W. M. Mitchell, Joseph F. Contrera, Synapse	1990	58
β-Endorphin-induced increases in plasma epinephrine, norepinephrine and dopamine in rats: inhibition of adrenomedullary response by intracerebral somatostatin Glen R. Van Loon, Nathan M. Appel, Doris Ho Brain Research MedicinePhysiologyβ-Endorphin-induced IncreasesNeuroendocrine MechanismNeuroendocrine Disorder	1981	58
Coexistence of serotonin- and substance P-like immunoreactivity in nerve fibers apposing identified sympathoadrenal preganglionic neurons in rat intermediolateral cell column Nathan M. Appel, Martin W. Wessendorf, Robert Elde Neuroscience Letters NeurophysiologyNeuroanatomySynaptic TransmissionPhysiologyNeurotransmitter	1986	56
Dopamine D3 Receptor Antagonist (GSK598809) Potentiates the Hypertensive Effects of Cocaine in Conscious, Freely-Moving Dogs Nathan M. Appel, Shou-Hua Li, Tyson H. Holmes, Journal of Pharmacology and Experimental Therapeutics	2015	47

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