J.E. Dumont

Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.

Jasmine Parma, J. Van Sande, S. Swillens et al. · Molecular Endocrinology · 1995 · 235 citations · Full text

Genetics, Molecular Biology, Regulatory Cascades +16

Anion Selectivity by the Sodium Iodide Symporter

J. Van Sande, C. Massart, Renaud Beauwens et al. · Endocrinology · 2002 · 172 citations · Full text

Human thyrotropin receptor gene: Expression in thyroid tumors and correlation to markers of thyroid differentiation and dedifferentiation

Georg Brabant, Carine Maenhaut, Josef Köhrle et al. · Molecular and Cellular Endocrinology · 1991 · 144 citations

Thyroid Tumors, Thyroid Disease, Pathology +7

Phosphorylation of mitogen-activated protein kinases is involved in the epidermal growth factor and phorbol ester, but not in the thyrotropin/cAMP, thyroid mitogenic pathway.

Françoise Lamy, Françoise Wilkin, Mireille Baptist et al. · Journal of Biological Chemistry · 1993 · 121 citations · Full text

Epidermal Growth Factor, Mitogen-activated Protein Kinases, Cell Proliferation +16

Selenium deficiency as a possible factor in the pathogenesis of myxoedematous endemic cretinism

Philippe Goyens, J. Golstein, B. Nsombola et al. · European Journal of Endocrinology · 1987 · 113 citations