John W. Tullai - Concepedia

Author

John W. Tullai

Boston University

Also Known As

John W Tullai, John W. Tullai

Publications

780

Citations

H-Index

Concepts

All Affiliations

Boston University Icahn School of Medicine at Mount Sinai University of Massachusetts Boston

About

John W. Tullai is an author at Boston University specializing in medicine, cell biology, and signal transduction.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	288
2	Cell Biology	6	6	288
3	Natural Sciences	3	3	216
4	Biochemistry	2	2	119
5	Cellular Physiology	2	2	119

John W. Tullai

Publications

777

Citations

H-Index

Page 1

	Year	Citations
Immediate-Early and Delayed Primary Response Genes Are Distinct in Function and Genomic Architecture John W. Tullai, Michael Schäffer, Steven Mullenbrock, Journal of Biological Chemistry	2007	221
Identification of Transcription Factor Binding Sites Upstream of Human Genes Regulated by the Phosphatidylinositol 3-Kinase and MEK/ERK Signaling Pathways John W. Tullai, Michael Schäffer, Steven Mullenbrock, Journal of Biological Chemistry Signaling PathwayCell RegulationReceptor Tyrosine KinaseAutophagyCellular Regulatory Mechanism	2004	97
Thiol Activation of Endopeptidase EC 3.4.24.15 Corie N. Shrimpton, Marc Glucksman, Rebecca A. Lew, Journal of Biological Chemistry	1997	83
Glycogen Synthase Kinase-3 Represses Cyclic AMP Response Element-binding Protein (CREB)-targeted Immediate Early Genes in Quiescent Cells John W. Tullai, Jie Chen, Michael Schäffer, Journal of Biological Chemistry	2007	72
The association of metalloendopeptidase EC 3.4.24.15 at the extracellular surface of the AtT-20 cell plasma membrane Peter J. Crack, T. John Wu, Philip M. Cummins, Brain Research BiochemistryMedicineMembrane TransportNatural SciencesMetalloprotein	1999	63
Secretion of Metalloendopeptidase 24.15 (EC 3.4.24.15) Emer S. Ferro, John W. Tullai, Marc Glucksman, DNA and Cell Biology Protein SecretionCellular PhysiologyOxidative StressEp24.15 ActivityBiosynthesis	1999	56
The Neuropeptide Processing Enzyme EC 3.4.24.15 Is Modulated by Protein Kinase A Phosphorylation John W. Tullai, Philip M. Cummins, Amanda Pabon, Journal of Biological Chemistry	2000	48
Phosphatidylinositol 3-kinase signaling in proliferating cells maintains an anti-apoptotic transcriptional program mediated by inhibition of FOXO and non-canonical activation of NFκB transcription factors Jolyon Terragni, Julie R. Graham, Kenneth W. Adams, BMC Cell Biology	2008	45
GSK-3 Represses Growth Factor-inducible Genes by Inhibiting NF-κB in Quiescent Cells Julie R. Graham, John W. Tullai, Geoffrey M. Cooper Journal of Biological Chemistry Gene InductionSignal TransductionSignaling PathwayCell RegulationImmunology	2009	32
A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells John W. Tullai, Julie R. Graham, Geoffrey M. Cooper Cell Cycle Immediate Early GenesCell DeathTranscriptional RegulationSignaling PathwayCell Regulation	2011	28

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