W N Hait - Concepedia

Author

W N Hait

Yale University

Also Known As

W Hait, W N Hait, W. Hait, W. N. Hait, W.N. Hait, William N. Hait, WilliamN. Hait, Z Lang

Publications

2.8K

Citations

H-Index

Concepts

All Affiliations

Yale University Johnson University Environmental and Occupational Health Sciences Institute Rutgers, The State University of New Jersey Rutgers Cancer Institute

About

W N Hait is an author at Yale University specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	20	785
2	Cell Biology	13	14	690
3	Health Sciences	5	5	115
4	Biochemistry	4	4	130
5	Natural Sciences	3	3	109

W N Hait

Publications

1.4K

Citations

H-Index

Page 1

	Year	Citations
Structural features determining activity of phenothiazines and related drugs for inhibition of cell growth and reversal of multidrug resistance. James M. Ford, Walter C. Prozialeck, W N Hait Molecular Pharmacology	1989	218
Biochemical correlates of the antitumor and antimitochondrial properties of gossypol enantiomers. Christopher C. Benz, Max A. Keniry, James M. Ford, Molecular Pharmacology	1990	112
DNA damage increases sensitivity to vinca alkaloids and decreases sensitivity to taxanes through p53-dependent repression of microtubule-associated protein 4. Christine C Zhang, Jin Yang, Judy Bash-Babula, PubMed Dna DamageChemoprevention StrategyMolecular RegulationGeneticsMolecular Biology	1999	100
Silencing of stathmin induces tumor-suppressor function in breast cancer cell lines harboring mutant p53 Elizabeth Alli, Jian‐Ming Yang, W N Hait Oncogene Mutant P53Breast OncologyMedicineBreast CancerTumor Suppressor	2006	97
Inhibition of growth of leukemic cells by inhibitors of calmodulin: Phenothiazines and melittin W N Hait, Linda Grais, Constance Benz, Cancer Chemotherapy and Pharmacology Medicinal ChemistryLeukemic CellsMedicinePharmacotherapyAnti-cancer Agent	1985	88
Activity and regulation by growth factors of calmodulin-dependent protein kinase III (elongation factor 2-kinase) in human breast cancer T. G. Parmer, Michael D. Ward, Edward J. Yurkow, British Journal of Cancer	1998	85
Disruption of the EF-2 kinase/Hsp90 protein complex: a possible mechanism to inhibit glioblastoma by geldanamycin. Jin Yang, Jin Yang, Marie A. Iannone, PubMed Tumor BiologyNeuro-oncologyGliomaMedicinePharmacology	2001	71
Effects of rottlerin, an inhibitor of calmodulin-dependent protein kinase III, on cellular proliferation, viability, and cell cycle distribution in malignant glioma cells. T. G. Parmer, Michael D. Ward, W N Hait PubMed Chemoprevention StrategyCam Kinase IiiCell DeathMalignant Glioma CellsGlioma	1997	64
Phosphorylation of elongation factor 2 in normal and malignant rat glial cells. D M Bagaglio, E H Cheng, Fred S. Gorelick, PubMed Cam Kinase IiiCell ProliferationGliomaCellular PhysiologyTumor Biology	1993	56
Phase I Clinical and Pharmacologic Study of 13-<i>cis</i>-Retinoic Acid, Interferon Alfa, and Paclitaxel in Patients With Prostate Cancer and Other Advanced Malignancies Robert S. DiPaola, Mohamed M. Rafi, Viral Vyas, Journal of Clinical Oncology	1999	50

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