Nibaldo C. Inestrosa

Author

Pontificia Universidad Católica de Chile

Also Known As

Inestrosa, N.C., N C Inestrosa, N. C. Inestrosa, N.C. Inestrosa, NIBALDO C. INESTROSA, Nibaldo C Inestrosa, Nibaldo C. INESTROSA, Nibaldo C. Inestrosa, Nibaldo Inestrosa

149

Publications

10.7K

Citations

H-Index

104

Concepts

All Affiliations

Pontificia Universidad Católica de Chile University of Chile UNSW Sydney Universidad de Magallanes

About

Nibaldo C. Inestrosa is an author at Pontificia Universidad Católica de Chile specializing in medicine, neuroscience, and alzheimer's disease.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	31	42	3K
2	Natural Sciences	8	8	638
3	Health Sciences	4	4	212
4	Engineering	2	2	68
5	Public Health	1	1	31

Nibaldo C. Inestrosa

Publications

7.9K

Citations

H-Index

Page 1

	Year	Citations
Acetylcholinesterase Accelerates Assembly of Amyloid-β-Peptides into Alzheimer's Fibrils: Possible Role of the Peripheral Site of the Enzyme Nibaldo C. Inestrosa, Alejandra Álvarez, Cristián A. Pérez, Neuron	1996	1.1K
Metalloenzyme-like Activity of Alzheimer's Disease β-Amyloid Carlos Opazo, Xudong Huang, Robert A. Cherny, Journal of Biological Chemistry	2002	574
A Structural Motif of Acetylcholinesterase That Promotes Amyloid β-Peptide Fibril Formation G. Ferrari, Mauricio Canales, Irina Shin, Biochemistry Protein ChemistryProteinlipid InteractionAcetylcholinesterase ThatStructural MotifSalt Concentration	2001	404
The α-Helical to β-Strand Transition in the Amino-terminal Fragment of the Amyloid β-Peptide Modulates Amyloid Formation Claudio Soto, Eduardo M. Castaño, Blas Frangione, Journal of Biological Chemistry	1995	320
Stable Complexes Involving Acetylcholinesterase and Amyloid-β Peptide Change the Biochemical Properties of the Enzyme and Increase the Neurotoxicity of Alzheimer’s Fibrils Alejandra Álvarez, Rodrigo Alarcón, Carlos Opazo, Journal of Neuroscience Amyloid-β Peptide ChangeNeurodegenerative DiseasesAlzheimer's DiseaseBiochemistryMedicine	1998	311
Activation of Wnt signaling by lithium and rosiglitazone reduced spatial memory impairment and neurodegeneration in brains of an APPswe/PSEN1ΔE9 mouse model of Alzheimer's disease Enrique M. Toledo, Nibaldo C. Inestrosa Molecular Psychiatry Alzheimer's DiseaseAppswe/psen1δe9 Mouse ModelDementiaSpatial Memory ImpairmentNeurochemical Biomarkers	2009	297
Wnt-3a overcomes β-amyloid toxicity in rat hippocampal neurons Alejandra Álvarez, Juan A. Godoy, Karin Müllendorff, Experimental Cell Research Neurodegenerative DiseasesMolecular NeuroscienceDegenerative PathologyRat Hippocampal NeuronsNeuroprotection	2004	213
β-Amyloid Causes Depletion of Synaptic Vesicles Leading to Neurotransmission Failure Jorge Parodí, Fernando J. Sepúlveda, Jorge Roa, Journal of Biological Chemistry	2009	176
Voluntary Running Attenuates Memory Loss, Decreases Neuropathological Changes and Induces Neurogenesis in a Mouse Model of <scp>A</scp>lzheimer's Disease Cheril Tapia‐Rojas, Florencia C. Aránguiz, Lorena Varela‐Nallar, Brain Pathology Neurochemical BiomarkersVoluntary ExerciseInduces NeurogenesisSynaptic SignalingTau Protein	2015	167
STI571 prevents apoptosis, tau phosphorylation and behavioural impairments induced by Alzheimer's β-amyloid deposits Gonzalo I. Cancino, Enrique M. Toledo, Nancy Leal, Brain	2008	144

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