Joep H.M. van Esch - Concepedia

Author

Joep H.M. van Esch

Also Known As

J H M van Esch, J. H. M. van Esch, J. H.M. van Esch, J.H.M. van Esch, Joep H M van Esch, Joep H. M. Esch, Joep H. M. van Esch, Joep H.M. Van Esch, Joep H.M. van Esch, Joep van Esch

Publications

2.3K

Citations

H-Index

Concepts

All Affiliations

Erasmus University Rotterdam Erasmus MC Hull York Medical School University Medical Center Utrecht Duke University

About

Joep H.M. van Esch is an author at Erasmus University Rotterdam specializing in pharmacology, medicine, and vascular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	11	11	557
2	Pharmacology	11	11	557
3	Nephrology	2	2	120
4	Diabetes	1	1	29
5	Endocrinology	1	1	58

Joep H.M. van Esch

Publications

1.3K

Citations

H-Index

Page 1

	Year	Citations
Hypertension Induced by the Tyrosine Kinase Inhibitor Sunitinib Is Associated With Increased Circulating Endothelin-1 Levels Mariëtte H.W. Kappers, Joep H.M. van Esch, Wim J. Sluiter, Hypertension	2010	281
The Vascular Endothelial Growth Factor Receptor Inhibitor Sunitinib Causes a Preeclampsia-Like Syndrome With Activation of the Endothelin System Mariëtte H.W. Kappers, Frank Smedts, T. Horn, Hypertension	2011	135
Sunitinib-Induced Hypothyroidism Is due to Induction of Type 3 Deiodinase Activity and Thyroidal Capillary Regression Mariëtte H.W. Kappers, Joep H.M. van Esch, Frank Smedts, The Journal of Clinical Endocrinology & Metabolism	2011	102
Treatment of Hypertension and Renal Injury Induced by the Angiogenesis Inhibitor Sunitinib Stephanie Lankhorst, Mariëtte H.W. Kappers, Joep H.M. van Esch, Hypertension HypertensionAngiogenesis InhibitionCardiovascular PharmacologyPharmacotherapyAngiogenesis	2014	91
Cardiac phenotype and angiotensin II levels in AT1a, AT1b, and AT2 receptor single, double, and triple knockouts Joep H.M. van Esch, Florian Gembardt, Anja Sterner‐Kock, Cardiovascular Research	2010	78
Selective Angiotensin-Converting Enzyme C-Domain Inhibition Is Sufficient to Prevent Angiotensin I–Induced Vasoconstriction Joep H.M. van Esch, Beril Tom, Vincent Dive, Hypertension HypertensionAngiotensin I–induced VasoconstrictionCardiovascular PharmacologyPharmacotherapyMolecular Pharmacology	2004	63
Compound 21 Induces Vasorelaxation via an Endothelium- and Angiotensin II Type 2 Receptor-Independent Mechanism Koen Verdonk, Matej Durik, Nalina Abd-Alla, Hypertension HypertensionCardiovascular PharmacologyCardiovascular FunctionBlood PressureMolecular Pharmacology	2012	58
Angiotensin II Type 2 Receptor– and Acetylcholine-Mediated Relaxation Bruno Sevá Pessôa, Denise E. Slump, Khatera Ibrahimi, Hypertension Molecular PharmacologyAcetylcholine-mediated RelaxationDevelopmental BiologyNitric OxideSex Hormones	2015	58
Effects of Angiotensin Metabolites in the Coronary Vascular Bed of the Spontaneously Hypertensive Rat Els Moltzer, Anna V.A. Verkuil, Richard van Veghel, Hypertension Coronary Vascular BedHypertensionAngiotensin MetabolitesCardiovascular DiseaseSpontaneously Hypertensive Rat	2009	57
Cardiovascular phenotype of mice lacking all three subtypes of angiotensin II receptors Florian Gembardt, Silvia Heringer‐Walther, Joep H.M. van Esch, The FASEB Journal HypertensionCardiovascular PharmacologyCardiovascular FunctionBlood PressureInflammation	2008	51

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