John M. Carney

Excess brain protein oxidation and enzyme dysfunction in normal aging and in Alzheimer disease.

Charles D. Smith, John M. Carney, Pamela Starke‐Reed et al. · Proceedings of the National Academy of Sciences · 1991 · 1.3K citations · Full text

Aging, Neurochemical Biomarkers, Creatine Kinase +16

A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease.

K. Hensley, John M. Carney, Mark P. Mattson et al. · Proceedings of the National Academy of Sciences · 1994 · 1.1K citations · Full text

Peptide Science, Beta-amyloid Aggregation, Redox Biology +18

Oxidative damage to brain proteins, loss of glutamine synthetase activity, and production of free radicals during ischemia/reperfusion-induced injury to gerbil brain.

Constance Oliver, Pamela Starke‐Reed, Earl R. Stadtman et al. · Proceedings of the National Academy of Sciences · 1990 · 778 citations · Full text

Lipid Peroxidation, Oxidative Damage, Redox Biology +21

Brain Regional Correspondence Between Alzheimer's Disease Histopathology and Biomarkers of Protein Oxidation

Kenneth Hensley, Nathan C. Hall, Subramaniam Ramachandran et al. · Journal of Neurochemistry · 1995 · 776 citations

Reversal of age-related increase in brain protein oxidation, decrease in enzyme activity, and loss in temporal and spatial memory by chronic administration of the spin-trapping compound N-tert-butyl-alpha-phenylnitrone.

John M. Carney, Pamela Starke‐Reed, Constance Oliver et al. · Proceedings of the National Academy of Sciences · 1991 · 724 citations · Full text

Chronic Administration, Lipid Peroxidation, Neurochemical Biomarkers +21