Concepedia

Author

Ian Sylvester

National Institutes of Health

Also Known As

I SYLVESTER, I Sylvester, I. F. Sylvester, I. Sylvester, Ian Sylvester

18

Publications

957

Citations

8

H-Index

22

Concepts

All Affiliations

National Institutes of HealthUniversity of Oxford
IS

About

Ian Sylvester is an author at National Institutes of Health specializing in cell biology, medicine, and gene expression.

Top Concepts

#ConceptH-IndexPublicationsCitations

1

Medicine

4

4

500

2

Cell Biology

4

4

500

3

Biochemistry

1

1

124

4

Immunology

1

1

124

5

Pharmacology

1

1

124

Ian Sylvester

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8

Publications

951

Citations

8

H-Index

YearCitations

Mouse Germline Restriction of Oct4 Expression by Germ Cell Nuclear Factor

Guy Fuhrmann, Arthur Chung, Kathy J. Jackson,

Developmental Cell

2001

252

Characterization of 2′-Fluoro-RNA Aptamers That Bind Preferentially to Disease-associated Conformations of Prion Protein and Inhibit Conversion

Alexandre Rhie, Louise Kirby, Natalie M. Sayer,

Journal of Biological Chemistry

2003

182

Structural Changes of the Prion Protein in Lipid Membranes Leading to Aggregation and Fibrillization

Jurate Kazlauskaite, Narinder Sanghera, Ian Sylvester,

Biochemistry

2003

158

Macrophages cultured in vitro release leukotriene B4 and neutrophil attractant/activation protein (interleukin 8) sequentially in response to stimulation with lipopolysaccharide and zymosan.

John A. Rankin, Ian Sylvester, Susan M. Smith,

Journal of Clinical Investigation

1990

124

Retinoic acid-mediated down-regulation of Oct3/4 coincides with the loss of promoter occupancy in vivo.

Saverio Minucci, V. Botquin, Young Il Yeom,

The EMBO Journal

1996

101

Regulation of the<i>Oct-4</i>gene by nuclear receptors

Ian Sylvester, Hans R. Schöler

Nucleic Acids Research

1994

88

Amyloid fibrils of mammalian prion protein induce axonal degeneration in NTERA2‐derived terminally differentiated neurons

Vera Novitskaya, Natallia Makarava, Ian Sylvester,

Journal of Neurochemistry

2007

23

Repression of Oct-4 during embryonic cell differentiation correlates with the appearance of TRIF, a transiently induced DNA-binding factor.

Gregor Fuhrmann, Ian Sylvester, Hans R. Schöler

PubMed

1999

23

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