S E Rittenhouse - Concepedia

Author

S E Rittenhouse

Also Known As

Parise, L. V., S E Rittenhouse, S. E. Rittenhouse, S.E. Rittenhouse, Susan E Rittenhouse, Susan E. RITTENHOUSE, Susan E. Rittenhouse, Susan Erika Rittenhouse

Publications

6.4K

Citations

H-Index

Concepts

All Affiliations

University of Vermont Thomas Jefferson University Brigham and Women's Hospital Sidney Kimmel Cancer Center Harvard University

About

S E Rittenhouse is an author at University of Vermont specializing in medicine, signal transduction, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	24	28	3.5K
2	Biochemistry	17	17	2.4K
3	Cell Biology	17	18	1.9K
4	Natural Sciences	14	14	2K
5	Health Sciences	3	3	539

S E Rittenhouse

Publications

5.3K

Citations

H-Index

Page 1

	Year	Citations
Sustained diacylglycerol formation from inositol phospholipids in angiotensin II-stimulated vascular smooth muscle cells. Kathy K. Griendling, S E Rittenhouse, T A Brock, Journal of Biological Chemistry Inositol PhospholipidsCardiovascular PharmacologyDiacylglycerol FormationCardiovascular FunctionCellular Physiology	1986	491
Mass changes in myoinositol trisphosphate in human platelets stimulated by thrombin. Inhibitory effects of phorbol ester. S E Rittenhouse, J P Sasson Journal of Biological Chemistry Cellular PhysiologyThrombosisHuman PlateletsHematologyPlatelet Antagonist	1985	293
Angiotensin increases inositol trisphosphate and calcium in vascular smooth muscle. R. Wayne Alexander, T A Brock, Michael A. Gimbrone, Hypertension HypertensionMuscle FunctionVascular Smooth MuscleCellular PhysiologyMuscle Physiology	1985	252
Cloning of a human phosphoinositide 3-kinase with a C2 domain that displays reduced sensitivity to the inhibitor wortmannin Jan Domin, Françoise Pagès, Stefano Volinia, Biochemical Journal	1997	250
Phorbol ester and 1-oleoyl-2-acetylglycerol inhibit angiotensin activation of phospholipase C in cultured vascular smooth muscle cells. T A Brock, S E Rittenhouse, Chris Powers, Journal of Biological Chemistry	1985	241
Regulation of the phosphoinositide hydrolysis pathway in thrombin-stimulated platelets by a pertussis toxin-sensitive guanine nucleotide-binding protein. Evaluation of its contribution to platelet activation and comparisons with the adenylate cyclase inhibitory protein, Gi. Lawrence F. Brass, Michael Laposata, H S Banga, Journal of Biological Chemistry Granule SecretionThrombin-stimulated PlateletsCellular PhysiologyThrombosisHematology	1986	232
Activation of phospholipases A and C in human platelets exposed to epinephrine: role of glycoproteins IIb/IIIa and dual role of epinephrine. H S Banga, Elizabeth R. Simons, L F Brass, Proceedings of the National Academy of Sciences Phospholipases APharmacotherapyInflammationThrombosisHuman Platelets	1986	228
Activation of platelet phosphatidylinositide 3-kinase requires the small GTP-binding protein Rho. Jin Zhang, W G King, Simon T. Dillon, Journal of Biological Chemistry	1993	211
Human platelets form 3-phosphorylated phosphoinositides in response to alpha-thrombin, U46619, or GTP gamma S. Gregory L. Kucera, S E Rittenhouse Journal of Biological Chemistry Molecular BiologyPolyphosphate Species PtdinsForm 3-Phosphorylated PhosphoinositidesCellular PhysiologyGtp-binding Protein	1990	202
Activated phosphoinositide 3-kinase associates with membrane skeleton in thrombin-exposed platelets. J Zhang, Michael Fry, Michael D. Waterfield, Journal of Biological Chemistry Molecular BiologyCytoskeletonActin PolymerizationMembrane SkeletonCellular Physiology	1992	184

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