Robert L. Reddick - Concepedia

Author

Robert L. Reddick

The University of Texas Health Science Center at San Antonio

Also Known As

Brinkhous, Kenneth, Griggs, Thomas, Maeda, Nobuyo, NICHOLS, T., R L Reddick, R REDDICK, R Reddick, R. L. Reddick, R. Reddick, R.L. Reddick

231

Publications

11.5K

Citations

H-Index

174

Concepts

All Affiliations

The University of Texas Health Science Center at San Antonio University of North Carolina at Chapel Hill South Texas Veterans Health Care System Murphy Oil Corporation (United States)The University of Texas at San Antonio

About

Robert L. Reddick is an author at The University of Texas Health Science Center at San Antonio specializing in medicine, immunology, and pathology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	37	70	7K
2	Health Sciences	13	13	3.4K
3	Public Health	3	3	157
4	Engineering	2	2	130
5	Natural Sciences	1	1	63

Robert L. Reddick

112

Publications

10.5K

Citations

H-Index

Page 1

	Year	Citations
Spontaneous Hypercholesterolemia and Arterial Lesions in Mice Lacking Apolipoprotein E Sunny H. Zhang, Robert L. Reddick, Jorge A. Piedrahita, Science Vascular DiseasePathologyHyperlipidemiaOxidative StressInflammation	1992	2.2K
Targeted Replacement of the Mouse Apolipoprotein E Gene with the Common Human APOE3 Allele Enhances Diet-induced Hypercholesterolemia and Atherosclerosis Patrick M. Sullivan, Hafid Mezdour, Yasuaki Aratani, Journal of Biological Chemistry	1997	583
Mice deficient in cystathionine beta-synthase: animal models for mild and severe homocyst(e)inemia. M. Watanabe, Jesús Osada, Yasuaki Aratani, Proceedings of the National Academy of Sciences Cell SpecializationMedicinePhysiologyImmunologyBlood Cell	1995	569
Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor A Paula M. Oliver, Jennifer E. Fox, Howard A. Rockman, Proceedings of the National Academy of Sciences HypertensionHeart FailureImmunologySudden DeathCardiovascular Function	1997	564
Enhanced atherosclerosis and kidney dysfunction in eNOS–/–Apoe–/– mice are ameliorated by enalapril treatment Joshua W. Knowles, Robert L. Reddick, J. Charles Jennette, Journal of Clinical Investigation	2000	300
Cc Chemokine Receptor (Ccr)2 Is Required for Langerhans Cell Migration and Localization of T Helper Cell Type 1 (Th1)-Inducing Dendritic Cells Naoko Sato, Sunil K. Ahuja, Marlon P. Quinones, The Journal of Experimental Medicine	2000	269
Aortic wall damage in mice unable to synthesize ascorbic acid Nobuyo Maeda, Hiroyuki Hagihara, Yukiko Nakata, Proceedings of the National Academy of Sciences	2000	258
A strategy to increase the donor pool: Use of cadaver lungs for transplantation Thomas M. Egan, Christopher Lambert, Robert L. Reddick, The Annals of Thoracic Surgery Lung TransplantationRegenerative MedicineDonor PoolTransplantationTranslational Research	1991	256
Estrogen receptor α is a major mediator of 17β-estradiol’s atheroprotective effects on lesion size in Apoe–/– mice Jeffrey B. Hodgin, John H. Krege, Robert L. Reddick, Journal of Clinical Investigation	2001	216
Lack of apoA-I is not associated with increased susceptibility to atherosclerosis in mice. Hao Li, Robert L. Reddick, Nobuo Maeda Arteriosclerosis and Thrombosis A Journal of Vascular Biology Vascular DiseaseGeneticsImmunologyApolipoprotein A-iHyperlipidemia	1993	191

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