Uwe‐Karsten Hanisch

Author

Uwe‐Karsten Hanisch

Also Known As

U Hanisch, U. Hanisch, U. K. Hanisch, U.-K Hanisch, U.-K. Hanisch, U.K Hanisch, UWE-KARSTEN HANISCH, Uwe-Karsten Hanisch, Uwe-Karsten Hanisch (251208), Uwe-karsten Hanisch

120

Publications

19.7K

Citations

H-Index

104

Concepts

All Affiliations

University of Göttingen Max Delbrück Center Universitätsmedizin Göttingen Evangelisches Krankenhaus Göttingen-Weende Charité - Universitätsmedizin Berlin

About

Uwe‐Karsten Hanisch is an author at University of Göttingen specializing in medicine, neuroinflammation, and neuroimmunology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	26	34	8K
2	Cell Biology	17	20	2.9K
3	Social Sciences	10	10	2.2K
4	Health Sciences	1	1	260
5	Anesthesia	1	1	92

Uwe‐Karsten Hanisch

Publications

12.9K

Citations

H-Index

Page 1

	Year	Citations
Microglia: active sensor and versatile effector cells in the normal and pathologic brain Uwe‐Karsten Hanisch, Helmut Kettenmann Nature Neuroscience InflammationActive SensorVersatile Effector CellsMedicineImmunology	2007	3.6K
Microglia in the adult brain arise from Ly-6ChiCCR2+ monocytes only under defined host conditions Alexander Mildner, Hauke Schmidt, Mirko Nitsche, Nature Neuroscience InflammationHost ConditionsNeuroregenerationMedicineAdult Brain Arise	2007	1.1K
Cell type– and brain region–resolved mouse brain proteome Kirti Sharma, Sebastian Schmitt, Caroline G. Bergner, Nature Neuroscience Cell BiologyNeuroscienceMolecular NeurobiologyBrain Proteome	2015	820
Selective transfer of exosomes from oligodendrocytes to microglia by macropinocytosis Dirk Fitzner, Mareike Schnaars, Denise van Rossum, Journal of Cell Science	2011	784
GFAP promoter-controlled EGFP-expressing transgenic mice: A tool to visualize astrocytes and astrogliosis in living brain tissue Christiané Nolte, Marina Matyash, Tatjyana Pivneva, Glia Living Brain TissueHighest Egfp ExpressionPeripheral NervesCellular NeurobiologyGlioma	2001	546
Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine Michael T. Heneka, Fabian Nadrigny, Tommy Regen, Proceedings of the National Academy of Sciences	2010	512
Microglial phenotype: is the commitment reversible? Michal Schwartz, Oleg Butovsky, Wolfgang Brück, Trends in Neurosciences MedicineBrain-immune InteractionNeurologyNeuroscienceNeuroimmunology	2006	420
Distinct and Nonredundant In Vivo Functions of IFNAR on Myeloid Cells Limit Autoimmunity in the Central Nervous System Marco Prinz, Hauke Schmidt, Alexander Mildner, Immunity InflammationAutoimmune DiseaseMedicineImmunologyVivo Functions	2008	383
c-Jun N-terminal kinases (JNKs) mediate pro-inflammatory actions of microglia Vicki Waetzig, K. Czeloth, Ute Hidding, Glia InflammationSignal TransductionC-jun N-terminal KinasesNuclear JnksMedicine	2005	290
Elevation of Basal Intracellular Calcium as a Central Element in the Activation of Brain Macrophages (Microglia): Suppression of Receptor-Evoked Calcium Signaling and Control of Release Function Anja Hoffmann, Oliver Kann, Carsten Ohlemeyer, Journal of Neuroscience Basal Intracellular CalciumBrain MacrophagesNitric OxideSynaptic TransmissionImmunology	2003	260

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