M S Nur-E-Kamal - Concepedia

Author

M S Nur-E-Kamal

Also Known As

M NurEKamal, M S A Nur-E-Kamal, M S Nur-E-Kamal, M S Nur-e-Kamal, M. S. A. Nur-E-Kamal, M. S. A. Nur-e-Kamal, M.S. A. NUR‐E‐KAMAL, M.S. Nur-E-Kamal, M.S.A. Nur-E-Kamal, M.S.A. Nur-e-Kamal

Publications

762

Citations

H-Index

Concepts

All Affiliations

Ludwig Cancer Research The Royal Melbourne Hospital United Arab Emirates University Ludwig Cancer Research Kyoto University

About

M S Nur-E-Kamal is an author at Ludwig Cancer Research specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	11	11	554
2	Cell Biology	10	10	473
3	Natural Sciences	5	5	189
4	Cellular Physiology	5	5	268
5	Biochemistry	1	1	21

M S Nur-E-Kamal

Publications

740

Citations

H-Index

Page 1

	Year	Citations
Molecular Cloning of a Novel Mitogen-Inducible Nuclear Protein with a Ran GTPase-Activating Domain That Affects Cell Cycle Progression Masakazu Hattori, Noriyuki Tsukamoto, M S Nur-E-Kamal, Molecular and Cellular Biology Ran Gtpase-activating DomainImmune RegulationImmunologyImmunologic MechanismCell Cycle	1995	91
Antibacterial activity of Euphorbia thymifolia Linn. Nawaz Khan, Mustafizur Rahman, M S Nur-E-Kamal PubMed MedicineAntibacterial ActivityPhytopharmacologyAntibacterial AgentMicrobiology	1988	81
Identification of a Neurite Outgrowth-Promoting Motif within the Alternatively Spliced Region of Human Tenascin-C M S Nur-E-Kamal, Mary Lynn T. Mercado Journal of Neuroscience Molecular BiologyCytoskeletonTenascin-c SequencesCell GrowthCellular Physiology	2001	64
The GTPase and Rho GAP domains of p190, a tumor suppressor protein that binds the M(r) 120,000 Ras GAP, independently function as anti-Ras tumor suppressors. D Z Wang, M S Nur-E-Kamal, Anjali Tikoo, PubMed Cancer ResearchRho Gap DomainsSystems BiologyCell RegulationRas Gap	1997	64
The role of Rho GTPases in the regulation of the rearrangement of actin cytoskeleton and cell movement Rokeya Begum, M S Nur-E-Kamal, M. A. Zaman Experimental & Molecular Medicine Cell AdhesionMolecular BiologyCytoskeletonCellular PhysiologyTumor Biology	2004	62
Molecular Characterization of Erythrocyte Glucose-6-Phosphate Dehydrogenase Deficiency in Al-Ain District, United Arab Emirates Riad Bayoumi, M S Nur-E-Kamal, M. Tadayyon, Human Heredity	1996	57
Asparagine 26, glutamic acid 31, valine 45, and tyrosine 64 of Ras proteins are required for their oncogenicity. M S Nur-E-Kamal, Andrew Sizeland, Giovanna M. D’Abaco, Journal of Biological Chemistry	1992	53
The CDC42-specific inhibitor derived from ACK-1 blocks v-Ha-Ras-induced transformation M S Nur-E-Kamal, Jabeen Kamal, M.M. Qureshi, Oncogene Molecular PharmacologySignal TransductionSignaling PathwayReceptor Tyrosine KinaseCdc42-specific Inhibitor	1999	50
The GTPase-activating NF1 fragment of 91 amino acids reverses v-Ha-Ras-induced malignant phenotype. M S Nur-E-Kamal, Mikael Varga, Hiroshi Maruta Journal of Biological Chemistry	1993	47
The minimal fragments of c-Raf-1 and NF1 that can suppress v-Ha-Ras-induced malignant phenotype. Masha Fridman, Anjali Tikoo, Mikael Varga, Journal of Biological Chemistry Ras-gtp ComplexImmunologyMinimal FragmentsCancer BiologyTumor Biology	1994	45

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