Jaime DeSantiago - Concepedia

Author

Jaime DeSantiago

University of Illinois Chicago

Also Known As

JAIME DESANTIAGO, Jaime DeSantiago, Jaime Desantiago

Publications

Citations

H-Index

Concepts

All Affiliations

University of Illinois Chicago Loyola University Chicago University of California, Davis University of Cincinnati University of California San Diego

About

Jaime DeSantiago is an author at University of Illinois Chicago specializing in physiology, medicine, and molecular physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	17	1K
2	Health Sciences	3	3	217
3	Natural Sciences	2	2	104
4	Biochemistry	2	2	104
5	Social Sciences	1	1	90

Jaime DeSantiago

Publications

Citations

H-Index

Page 1

	Year	Citations
Transgenic CaMKIIδ <sub>C</sub> Overexpression Uniquely Alters Cardiac Myocyte Ca <sup>2+</sup> Handling Lars S. Maier, Tong Zhang, Lu Chen, Circulation Research	2003	428
Phosphorylation of phospholamban and troponin I in β-adrenergic-induced acceleration of cardiac relaxation Li Li, Jaime DeSantiago, Guoxiang Chu, American Journal of Physiology-Heart and Circulatory Physiology	2000	236
Frequency-dependent Acceleration of Relaxation in the Heart Depends on CaMKII, but not Phospholamban Jaime DeSantiago, Lars S. Maier, Donald M. Bers Journal of Molecular and Cellular Cardiology Cardiac MuscleHeart FailureKinesiologyCardiac MechanicPhysiology	2002	169
Cardiac Alternans Do Not Rely on Diastolic Sarcoplasmic Reticulum Calcium Content Fluctuations Eckard Picht, Jaime DeSantiago, Lothar A. Blatter, Circulation Research Cardiac MuscleCardiac Alternans DoHeart FailureSignal TransductionMolecular Physiology	2006	168
The IP <sub>3</sub> Receptor Regulates Cardiac Hypertrophy in Response to Select Stimuli Hiroyuki Nakayama, Ilona Bódi, Marjorie Maillet, Circulation Research	2010	168
Arrhythmogenic Effects of β <sub>2</sub> -Adrenergic Stimulation in the Failing Heart Are Attributable to Enhanced Sarcoplasmic Reticulum Ca Load Jaime DeSantiago, Xun Ai, Mohammed A. Islam, Circulation Research Cardiac MuscleArrhythmogenic EffectsHeart FailureMolecular PhysiologyCardiomyopathy	2008	108
Akt regulates L-type Ca2+ channel activity by modulating Cavα1 protein stability Daniele Catalucci, Deng-Hong Zhang, Jaime DeSantiago, The Journal of Cell Biology Cardiac MuscleMolecular RegulationCellular PhysiologyInsulin SignalingTranscriptional Regulation	2009	107
Akt regulates L-type Ca2+ channel activity by modulating Cavα1 protein stability Daniele Catalucci, Deng-Hong Zhang, Jaime DeSantiago, The Journal of Cell Biology Cardiac MuscleMolecular RegulationCellular PhysiologySignaling PathwayLtcc Stability	2013	90
CaMKII inhibition targeted to the sarcoplasmic reticulum inhibits frequency-dependent acceleration of relaxation and Ca2+ current facilitation Eckard Picht, Jaime DeSantiago, Sabine Huke, Journal of Molecular and Cellular Cardiology Cell PhysiologySarcoplasmic ReticulumSignal TransductionMolecular PhysiologyBiochemistry	2006	90
The inotropic effect of cardioactive glycosides in ventricular myocytes requires Na<sup>+</sup>–Ca<sup>2</sup><sup>+</sup> exchanger function Julio Altamirano, Yanxia Li, Jaime DeSantiago, The Journal of Physiology	2006	86

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