Shantie Jagmohan–Changur

Author

Also Known As

Daniel Barker, Shantie C. Jagmohan-Changur, Shantie C. Jagmohan‐Changur, Shantie Jagmohan-Changur, Shantie Jagmohan‐Changur, Shantie Jagmohan–Changur

Publications

899

Citations

H-Index

Concepts

All Affiliations

Leiden University Medical Center Leiden University Netherlands Consortium for Healthy Ageing University Medical Center Utrecht Institute of Cancer Research

About

Shantie Jagmohan–Changur is an author at Leiden University Medical Center specializing in medicine, pathology, and cancer research.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	590
2	Pathology	7	7	590
3	Cell Biology	2	2	188
4	Gastroenterology	1	1	54
5	Molecular Genetics	1	1	46

Shantie Jagmohan–Changur

Publications

868

Citations

H-Index

Page 1

	Year	Citations
Heterozygous Mutations in PMS2 Cause Hereditary Nonpolyposis Colorectal Carcinoma (Lynch Syndrome) Yvonne Hendriks, Shantie Jagmohan–Changur, Heleen M. van der Klift, Gastroenterology Lynch SyndromeGenetic DisorderGeneticsColorectal CancerPathology	2006	153
Apc1638N: A mouse model for familial adenomatous polyposis–associated desmoid tumors and cutaneous cysts Ron Smits, Willemien van der Houven van Oordt, Arne Luz, Gastroenterology Tumor BiologyTumoral PathologyCutaneous CystsDesmoid TumorsMedicine	1998	134
Cyclooxygenase-two (COX-2) modulates proliferation in aggressive fibromatosis (desmoid tumor) Raymond Poon, Ron Smits, Catherine Li, Oncogene Tumor BiologyDesmoid TumorFibrosisPathologyMedicine	2001	106
SomaticApc mutations are selected upon their capacity to inactivate the ?-catenin downregulating activity Ron Smits, Nandy Hofland, Winfried Edelmann, Genes Chromosomes and Cancer	2000	91
EXO1 variants occur commonly in normal population: evidence against a role in hereditary nonpolyposis colorectal cancer. Shantie Jagmohan–Changur, Taija Poikonen, Susa Vilkki, PubMed Genome InstabilityEuropean Crc PatientsCancer RiskMismatch Repair GenesGastrointestinal Oncology	2003	68
Enrichment of Low Penetrance Susceptibility Loci in a Dutch Familial Colorectal Cancer Cohort Anneke Middeldorp, Shantie Jagmohan–Changur, Ronald van Eijk, Cancer Epidemiology Biomarkers & Prevention	2009	68
E-cadherin and adenomatous polyposis coli mutations are synergistic in intestinal tumor initiation in mice Ron Smits, Patricia Ruíz, Salvador J. Díaz‐Cano, Gastroenterology MedicineGastroenterologyPathologyColorectal CancerGastrointestinal Pathology	2000	54
Serrated adenomas and mixed polyposis caused by a splice acceptor deletion in the mouse <i>Smad4</i> gene Peter Hohenstein, Lia Molenaar, Joyce Elsinga, Genes Chromosomes and Cancer Somatic VariantTumor HeterogeneitySplice Site MutationGeneticsSplice Acceptor Deletion	2003	46
Combined analysis of three lynch syndrome cohorts confirms the modifying effects of 8q23.3 and 11q23.1 in MLH1 mutation carriers Bente A. Talseth‐Palmer, Juul Wijnen, Ingvild Synnøve Brenne, International Journal of Cancer	2012	35
Genomic acute myeloid leukemia-associated inv(16)(p13q22) breakpoints are tightly clustered Bert A. van der Reijden, Hans G. Dauwerse, Rachel H. Giles, Oncogene Hematological MalignancyChromatinMixed-phenotype Acute LeukemiaHematologyPathology	1999	29

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