J. Stefan Kaczmarek - Concepedia

Author

J. Stefan Kaczmarek

Howard Hughes Medical Institute

Also Known As

J. Kaczmarek, J. S. Kaczmarek, J. Stefan Kaczmarek, Jan Stefan Kaczmarek

Publications

966

Citations

H-Index

Concepts

All Affiliations

Howard Hughes Medical Institute Harvard University Friedrich-Alexander-Universität Erlangen-Nürnberg

About

J. Stefan Kaczmarek is an author at Howard Hughes Medical Institute specializing in medicine, cellular physiology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	4	4	731
2	Social Sciences	2	2	428
3	Cell Biology	2	2	303
4	Natural Sciences	1	1	47
5	Biochemistry	1	1	256

J. Stefan Kaczmarek

Publications

954

Citations

H-Index

Page 1

	Year	Citations
Hv1 proton channels are required for high-level NADPH oxidase-dependent superoxide production during the phagocyte respiratory burst I. Scott Ramsey, Evelyne Ruchti, J. Stefan Kaczmarek, Proceedings of the National Academy of Sciences Hv1 Proton ChannelsMicrobial PathogensInnate Immune SystemImmunologyBacterial Pathogens	2009	256
Transient receptor potential cation channel, subfamily C, member 5 (TRPC5) is a cold-transducer in the peripheral nervous system Katharina Zimmermann, Jochen K. Lennerz, Alexander Hein, Proceedings of the National Academy of Sciences NeurotransmitterMolecular PainNeurotransmissionPeripheral NerveMember 5	2011	254
Phenotyping sensory nerve endings in vitro in the mouse Katharina Zimmermann, Alexander Hein, Ulrich Hager, Nature Protocols Developmental BiologyPeripheral Nerve InjuryNeurophysiologyNeuroanatomyNeuroscience	2009	174
Intracellular calcium strongly potentiates agonist-activated TRPC5 channels Nathaniel T. Blair, J. Stefan Kaczmarek, David E. Clapham The Journal of General Physiology	2009	152
Calpain cleaves and activates the TRPC5 channel to participate in semaphorin 3A-induced neuronal growth cone collapse J. Stefan Kaczmarek, Antonio Riccio, David E. Clapham Proceedings of the National Academy of Sciences	2012	71
TATA-Binding Protein (TBP)-Like Factor (TLF) Is a Functional Regulator of Transcription: Reciprocal Regulation of the Neurofibromatosis Type 1 and c-<i>fos</i> Genes by TLF/TRF2 and TBP Jayhong A. Chong, Magdalene M. Moran, Martin Teichmann, Molecular and Cellular Biology Molecular RegulationSynaptic SignalingTranscriptional RegulationSignaling PathwayCellular Regulatory Mechanism	2005	47

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