Mohammed A. Nassar - Concepedia

Author

Mohammed A. Nassar

Also Known As

M A Nassar, M. A. Nassar, Mohammed A Nassar, Mohammed A. Nassar, Mohammed Abd-Ellatif Nassar, Mohammed Nassar, Mohammed A. Nassar, Nassar, Mohammed A.

Publications

4.9K

Citations

H-Index

Concepts

All Affiliations

University College London University of Sheffield King's College London Seoul National University Queen Mary University of London

About

Mohammed A. Nassar is an author at University College London specializing in medicine, neuroscience, and nervous system.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	11	11	1.8K
2	Social Sciences	5	5	681
3	Health Sciences	4	4	826
4	Natural Sciences	1	1	141
5	Biochemistry	1	1	141

Mohammed A. Nassar

Publications

4.8K

Citations

H-Index

Page 1

	Year	Citations
Nociceptor-specific gene deletion reveals a major role for Na<sub>v</sub>1.7 (PN1) in acute and inflammatory pain Mohammed A. Nassar, L Caroline Stirling, Greta Forlani, Proceedings of the National Academy of Sciences	2004	676
The Cell and Molecular Basis of Mechanical, Cold, and Inflammatory Pain Bjarke Abrahamsen, Jing Zhao, Curtis O. Asante, Science Pain DisordersAcute PainPain MedicineImmunologyNeuropathic Pain	2008	469
Parallel “Pain” Pathways Arise from Subpopulations of Primary Afferent Nociceptor Joao Bráz, Mohammed A. Nassar, John N. Wood, Neuron Pain ResearchPain DisordersPain SyndromeHealth SciencesAcute Pain	2005	299
A P2X purinoceptor cDNA conferring a novel pharmacological profile Xuenong Bo, Yi Zhang, Mohammed A. Nassar, FEBS Letters	1995	280
Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons Michael S. Minett, Mohammed A. Nassar, Anna K. Clark, Nature Communications	2012	275
Nociceptor-specific gene deletion using heterozygous NaV1.8-Cre recombinase mice Caroline L. Stirling, Greta Forlani, Mark D. Baker, Pain Molecular PainNeurotransmissionSocial SciencesNav1.8 PromoterNociceptor-specific Gene Deletion	2004	261
Neuropathic Pain Develops Normally in Mice Lacking both Na<sub>v</sub>1.7 and Na<sub>v</sub>1.8 Mohammed A. Nassar, Alessandra Levato, L Caroline Stirling, Molecular Pain	2005	204
Pain without Nociceptors? Nav1.7-Independent Pain Mechanisms Michael S. Minett, Sarah Falk, Sonia Santana‐Varela, Cell Reports Pain MedicineAnesthetic MechanismNeuropathic PainMolecular PainNav1.7 Sodium Channels	2014	169
Small RNAs Control Sodium Channel Expression, Nociceptor Excitability, and Pain Thresholds Jing Zhao, Man-Cheung Lee, Ali Momin, Journal of Neuroscience	2010	164
Nociceptor-derived brain-derived neurotrophic factor regulates acute and inflammatory but not neuropathic pain Jing Zhao, Anjan Seereeram, Mohammed A. Nassar, Molecular and Cellular Neuroscience InflammationNeuroinflammationPain SyndromePain MedicineMedicine	2006	163

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